CWD(Chronic Wasting Disease) Meeting

[BuxtonHunter]

I recieved this email earlier today and thought I'd put it on here as the quickest way I know to inform fellow whitetail hunters about this meeting. It may already be on here and if it is I apologize.

As we discussed earlier, there is a CWD(Chronic Wasting Disease) Meeting, called by the legislators, scheduled for this Tuesday night, October 2nd,2012, at 7:00PM, to be located at the-UFCW Hall, 1301 E. Marys St., Ottumwa, Iowa (This hall is located on the SE corner of Ottumwa on E. Marys St and just West of Hwy 63).



All concerned Press, Sportsman or anyone interested in the Outcome of Iowa’s Deer herd, should attend!! Please, spread the word!



CWD has now been found in Pens, at three locations in Iowa-Davis Co., Cerro Gordo Co., and now Pottawattamie Co. –It appears, all three, are a result of deer transferred from a breeding pen owned by Tom Brakke in Cerro Gordo Co., Ia. It also appears there are several other pens, around Iowa, that are under quarantine by the Dept. of Ag., that have deer from Brakke’s pen.



Unfortunately, Politics rather than Science, may manage this important case of CWD in Iowa. Why isn’t the existing CWD Containment Plan going to be followed?

It appears the Governor’s Office is directing the CWD process. Why not the DNR? We have heard that Doug Gross an attorney, former candidate for Governor, and formerly Gov. Branstads Chief of Staff, is the attorney representing Tom Brakke, the CWD pen owner.

Following is a link, to an informative article on CWD in Iowa - http://thegazette.com/2012/09/21/disease-raises-concerns-about-deer-farms-in-iowa/
Thanks for your concern!

 

[loneranger]

Money $$$$ talks. All we need. The midges,,CWD,,high number of doe tags.There will be no deer left. DNR should limit tags,,and seasons. Oh,,I forgot $$$$.

 

[6x6]

Money $$$$ talks. All we need. The midges,,CWD,,high number of doe tags.There will be no deer left. DNR should limit tags,,and seasons. Oh,,I forgot $$$$.

X2

It is odd that in the story the preserve owner says he has been in the cwd monitoring program for 10 years not bringing in any new deer, only raising his own stock. Most only buy semen for new bloodlines. So how did his herd become exposed to it then if thats the truth.

 

[flounder9]

re-CWD (Chronic Wasting Disease) Meeting

re-CWD (Chronic Wasting Disease) Meeting...

GOOD LUCK !!!


PLEASE SEE ;




page 30,



*** Spraker suggested an interesting explanation for the occurrence of CWD. The deer pens at the Foot Hills Campus were built some 30-40 years ago by a Dr. Bob Davis.

At or about that time, allegedly, some scrapie work was conducted at this site. When deer were introduced to the pens they occupied ground that had previously been occupied by sheep.




(PLEASE NOTE SOME OF THESE OLD UK GOVERNMENT FILE URLS ARE SLOW TO OPEN, AND SOMETIMES YOU MAY HAVE TO CLICK ON MULTIPLE TIMES, PLEASE BE PATIENT, ANY PROBLEMS
PLEASE WRITE ME PRIVATELY, AND I WILL TRY AND FIX OR SEND YOU OLD PDF FILE...TSS)




IN CONFIDENCE


PERCEPTIONS OF UNCONVENTIONAL SLOW VIRUS DISEASES OF ANIMALS IN USA


GAH WELLS


REPORT OF A VISIT TO THE USA APRIL-MAY 1989




http://collections.europarchive.org...www.bseinquiry.gov.uk/files/mb/m11b/tab01.pdf




now, years later, see the latest studies here on scrapie and cwd ;



PO-039: A comparison of scrapie and chronic wasting disease in white-tailed deer


Justin Greenlee, Jodi Smith, Eric Nicholson US Dept. Agriculture; Agricultural Research Service, National Animal Disease Center; Ames, IA USA


Interspecies transmission studies afford the opportunity to better understand the potential host range and origins of prion diseases. The purpose of these experiments was to determine susceptibility of white-tailed deer (WTD) to scrapie and to compare the resultant clinical signs, lesions, and molecular profiles of PrPSc to those of chronic wasting disease (CWD). We inoculated WTD intracranially (IC; n = 5) and by a natural route of exposure (concurrent oral and intranasal (IN); n = 5) with a US scrapie isolate. All deer were inoculated with a 10% (wt/vol) brain homogenate from sheep with scrapie (1ml IC, 1 ml IN, 30 ml oral). All deer inoculated by the intracranial route had evidence of PrPSc accumulation. PrPSc was detected in lymphoid tissues as early as 7 months-post-inoculation (PI) and a single deer that was necropsied at 15.6 months had widespread distribution of PrPSc highlighting that PrPSc is widely distributed in the CNS and lymphoid tissues prior to the onset of clinical signs. IC inoculated deer necropsied after 20 months PI (3/5) had clinical signs, spongiform encephalopathy, and widespread distribution of PrPSc in neural and lymphoid tissues. The results of this study suggest that there are many similarities in the manifestation of CWD and scrapie in WTD after IC inoculation including early and widespread presence of PrPSc in lymphoid tissues, clinical signs of depression and weight loss progressing to wasting, and an incubation time of 21-23 months. Moreover, western blots (WB) done on brain material from the obex region have a molecular profile similar to CWD and distinct from tissues of the cerebrum or the scrapie inoculum. However, results of microscopic and IHC examination indicate that there are differences between the lesions expected in CWD and those that occur in deer with scrapie: amyloid plaques were not noted in any sections of brain examined from these deer and the pattern of immunoreactivity by IHC was diffuse rather than plaque-like. After a natural route of exposure, 100% of WTD were susceptible to scrapie. Deer developed clinical signs of wasting and mental depression and were necropsied from 28 to 33 months PI. Tissues from these deer were positive for PrPSc by IHC and WB. Similar to IC inoculated deer, samples from these deer exhibited two different molecular profiles: samples from obex resembled CWD whereas those from cerebrum were similar to the original scrapie inoculum. On further examination by WB using a panel of antibodies, the tissues from deer with scrapie exhibit properties differing from tissues either from sheep with scrapie or WTD with CWD. Samples from WTD with CWD or sheep with scrapie are strongly immunoreactive when probed with mAb P4, however, samples from WTD with scrapie are only weakly immunoreactive. In contrast, when probed with mAb’s 6H4 or SAF 84, samples from sheep with scrapie and WTD with CWD are weakly immunoreactive and samples from WTD with scrapie are strongly positive. This work demonstrates that WTD are highly susceptible to sheep scrapie, but on first passage, scrapie in WTD is differentiable from CWD.


http://www.landesbioscience.com/journals/prion/03-Prion6-2-Transmission-and-strains.pdf



White-tailed Deer are Susceptible to Scrapie by Natural Route of Infection


Jodi D. Smith, Justin J. Greenlee, and Robert A. Kunkle; Virus and Prion Research Unit, National Animal Disease Center, USDA-ARS


Interspecies transmission studies afford the opportunity to better understand the potential host range and origins of prion diseases. Previous experiments demonstrated that white-tailed deer are susceptible to sheep-derived scrapie by intracranial inoculation. The purpose of this study was to determine susceptibility of white-tailed deer to scrapie after a natural route of exposure. Deer (n=5) were inoculated by concurrent oral (30 ml) and intranasal (1 ml) instillation of a 10% (wt/vol) brain homogenate derived from a sheep clinically affected with scrapie. Non-inoculated deer were maintained as negative controls. All deer were observed daily for clinical signs. Deer were euthanized and necropsied when neurologic disease was evident, and tissues were examined for abnormal prion protein (PrPSc) by immunohistochemistry (IHC) and western blot (WB). One animal was euthanized 15 months post-inoculation (MPI) due to an injury. At that time, examination of obex and lymphoid tissues by IHC was positive, but WB of obex and colliculus were negative. Remaining deer developed clinical signs of wasting and mental depression and were necropsied from 28 to 33 MPI. Tissues from these deer were positive for scrapie by IHC and WB. Tissues with PrPSc immunoreactivity included brain, tonsil, retropharyngeal and mesenteric lymph nodes, hemal node, Peyer’s patches, and spleen. This work demonstrates for the first time that white-tailed deer are susceptible to sheep scrapie by potential natural routes of inoculation. In-depth analysis of tissues will be done to determine similarities between scrapie in deer after intracranial and oral/intranasal inoculation and chronic wasting disease resulting from similar routes of inoculation.



see full text ;



http://www.usaha.org/Portals/6/Reports/2010/report-cwal-2010.pdf





UPDATE OCTOBER 2012 TSE CWD




a bit of other data on cwd here you may be interested in ;





see game farm high fence information next two links;




*** Friday, September 21, 2012

Chronic Wasting Disease CWD raises concerns about deer farms in Iowa

http://chronic-wasting-disease.blogspot.com/2012/09/chronic-wasting-disease-cwd-raises.html






*** Friday, September 28, 2012

Stray elk renews concerns about deer farm security Minnesota

http://chronic-wasting-disease.blogspot.com/2012/09/stray-elk-renews-concerns-about-deer.html






Friday, August 24, 2012

Diagnostic accuracy of rectal mucosa biopsy testing for chronic wasting disease within white-tailed deer (Odocoileus virginianus) herds in North America

http://chronic-wasting-disease.blogspot.com/2012/08/diagnostic-accuracy-of-rectal-mucosa.html






Saturday, September 01, 2012

Resistance of Soil-Bound Prions to Rumen Digestion

http://chronic-wasting-disease.blogspot.com/2012/09/resistance-of-soil-bound-prions-to.html






Monday, September 17, 2012

Rapid Transepithelial Transport of Prions Following Inhalation

http://transmissiblespongiformencep...12/09/rapid-transepithelial-transport-of.html





Saturday, February 04, 2012

Wisconsin 16 MONTH age limit on testing dead deer Game Farm CWD Testing Protocol Needs To Be Revised

http://chronic-wasting-disease.blogspot.com/2012/02/wisconsin-16-age-limit-on-testing-dead.html




Monday, June 11, 2012

OHIO Captive deer escapees and non-reporting

http://chronic-wasting-disease.blogspot.com/2012/06/ohio-captive-deer-escapees-and-non.html




Tuesday, September 11, 2012


Agreement Reached with Owner to De-Populate CWD Deer at Davis County Hunting Preserve Iowa


http://chronic-wasting-disease.blogspot.com/2012/09/agreement-reached-with-owner-to-de.html




Wednesday, September 05, 2012


Additional Facility in Pottawatamie County Iowa Under Quarantine for CWD after 5 deer test positive


http://chronic-wasting-disease.blogspot.com/2012/09/additional-facility-in-pottawatamie.html




Friday, July 20, 2012


CWD found for first time in Iowa at hunting preserve


http://chronic-wasting-disease.blogspot.com/2012/07/cwd-found-for-first-time-in-iowa-at.html




Tuesday, June 05, 2012

Captive Deer Breeding Legislation Overwhelmingly Defeated During 2012 Legislative Session

http://chronic-wasting-disease.blogspot.com/2012/06/captive-deer-breeding-legislation.html






GAME FARMERS, CWD, AND THEIR COMMENTS...disturbing...frightening even. it seems they are oblivious to their own demise. ...


see comments ;


http://www.regulations.gov/#!docketDetail;rpp=250;so=ASC;sb=postedDate;po=300;D=APHIS-2006-0118




CWD has been identified in free-ranging cervids in 15 US states and 2 Canadian provinces and in ≈ 100 captive herds in 15 states and provinces and in South Korea (Figure 1, panel B). SNIP... Long-term effects of CWD on cervid populations and ecosystems remain unclear as the disease continues to spread and prevalence increases. In captive herds, CWD might persist at high levels and lead to complete herd destruction in the absence of human culling. Epidemiologic modeling suggests the disease could have severe effects on free-ranging deer populations, depending on hunting policies and environmental persistence (8,9). CWD has been associated with large decreases in free-ranging mule deer populations in an area of high CWD prevalence (Boulder, Colorado, USA) (5).

PLEASE STUDY THIS MAP, COMPARE FARMED CWD TO WILD CWD...TSS



http://wwwnc.cdc.gov/eid/article/18/3/11-0685-f1.htm




Saturday, February 18, 2012

Occurrence, Transmission, and Zoonotic Potential of Chronic Wasting Disease

CDC Volume 18, Number 3—March 2012

CWD has been identified in free-ranging cervids in 15 US states and 2 Canadian provinces and in ≈100 captive herds in 15 states and provinces and in South Korea (Figure 1, panel B).



http://wwwnc.cdc.gov/eid/article/18/3/11-0685_article.htm







Thursday, February 09, 2012


50 GAME FARMS IN USA INFECTED WITH CHRONIC WASTING DISEASE


http://chronic-wasting-disease.blogspot.com/2012/02/50-game-farms-to-date-in-usa-infected.html




Tuesday, December 20, 2011

CHRONIC WASTING DISEASE CWD WISCONSIN Almond Deer (Buckhorn Flats) Farm Update DECEMBER 2011

The CWD infection rate was nearly 80%, the highest ever in a North American captive herd.

RECOMMENDATION: That the Board approve the purchase of 80 acres of land for $465,000 for the Statewide Wildlife Habitat Program in Portage County and approve the restrictions on public use of the site.

snip...see full text and much more here ;



http://dnr.wi.gov/org/nrboard/2011/december/12-11-2b2.pdf




2010 WISCONSIN CAPTIVE DEER ESCAPES




> There were 26 reported escape incidents so far this year, this amounted to 20 actual confirmed escape incidents because 3 were previously reported, 2 were confirmed as wild deer, and 1 incident was not confirmed.


Wisconsin Conservation Congress CWD Committee Notes recorded by Secretary- Tony Grabski, Iowa County Delegate From the meeting at Mead Wildlife Area Visitor Center Milladore, WI Saturday, August 7, 2010, 9:30 AM

C. & D. Captive Cervid and Law Enforcement Update (11:10 AM)- Warden Pete Dunn gave the captive cervid farm update. There were 26 reported escape incidents so far this year, this amounted to 20 actual confirmed escape incidents because 3 were previously reported, 2 were confirmed as wild deer, and 1 incident was not confirmed. Approximately 30% of these escapes were caused by gates being left open and the other 70% resulted from bad fencing or fence related issues. The 20 actual confirmed escape incidents amounted to 77 total animals. 50 of the escaped animals were recovered or killed and 27 were not recovered and remain unaccounted for. Last year the CWD Committee passed a resolution to require double gates, but this has not gone into effect yet. Questions were raised by the committee about double fencing requirements? Pete responded that double fencing has not been practical or accepted by the industry. The DNR has the authority to do fence inspections. ?If a fence fails to pass the inspection the fencing certificate can be revoked and the farmer can be issued a citation. This year three citations and one warning have been issued for escapes.


http://dnr.wi.gov/org/nrboard/congress/minutes/2010/cwd_committee_2010.pdf




and just for the record, the above 2010 report and statement there from i.e. ;




> Tami Ryan agreed and added that the risk of transmission through water was low because prions bind to soils preferentially.



this needs to be addressed, because risk factor for water from cwd endemic areas is a serious risk factor in my opinion. please see ;



Detection of Protease-Resistant Prion Protein in Water from a CWD-Endemic Area

snip...

These data suggest prolonged persistence and accumulation of prions in the environment that may promote CWD transmission.

snip...

The data presented here demonstrate that sPMCA can detect low levels of PrPCWD in the environment, corroborate previous biological and experimental data suggesting long term persistence of prions in the environment2,3 and imply that PrPCWD accumulation over time may contribute to transmission of CWD in areas where it has been endemic for decades. This work demonstrates the utility of sPMCA to evaluate other environmental water sources for PrPCWD, including smaller bodies of water such as vernal pools and wallows, where large numbers of cervids congregate and into which prions from infected animals may be shed and concentrated to infectious levels.

snip...end...full text at ;




http://www.landesbioscience.com/



http://www.cwd-info.org/pdf/3rd_CWD_Symposium_utah.pdf



http://chronic-wasting-disease.blogspot.com/2009/08/third-international-cwd-symposium-july.html



http://chronic-wasting-disease.blogspot.com/2009/10/detection-of-protease-resistant-cervid.html







http://chronic-wasting-disease.blogspot.com/






LANCET INFECTIOUS DISEASE JOURNAL

Volume 3, Number 8 01 August 2003

Newsdesk

Tracking spongiform encephalopathies in North America

Xavier Bosch

My name is Terry S Singeltary Sr, and I live in Bacliff, Texas. I lost my mom to hvCJD (Heidenhain variant CJD) and have been searching for answers ever since. What I have found is that we have not been told the truth. CWD in deer and elk is a small portion of a much bigger problem.

49-year-old Singeltary is one of a number of people who have remained largely unsatisfied after being told that a close relative died from a rapidly progressive dementia compatible with spontaneous Creutzfeldt-Jakob disease (CJD). So he decided to gather hundreds of documents on transmissible spongiform encephalopathies (TSE) and realised that if Britons could get variant CJD from bovine spongiform encephalopathy (BSE), Americans might get a similar disorder from chronic wasting disease (CWD) the relative of mad cow disease seen among deer and elk in the USA. Although his feverish search did not lead him to the smoking gun linking CWD to a similar disease in North American people, it did uncover a largely disappointing situation.

Singeltary was greatly demoralised at the few attempts to monitor the occurrence of CJD and CWD in the USA. Only a few states have made CJD reportable. Human and animal TSEs should be reportable nationwide and internationally, he complained in a letter to the Journal of the American Medical Association (JAMA 2003; 285: 733). I hope that the CDC does not continue to expect us to still believe that the 85% plus of all CJD cases which are sporadic are all spontaneous, without route or source.

Until recently, CWD was thought to be confined to the wild in a small region in Colorado. But since early 2002, it has been reported in other areas, including Wisconsin, South Dakota, and the Canadian province of Saskatchewan. Indeed, the occurrence of CWD in states that were not endemic previously increased concern about a widespread outbreak and possible transmission to people and cattle.

To date, experimental studies have proven that the CWD agent can be transmitted to cattle by intracerebral inoculation and that it can cross the mucous membranes of the digestive tract to initiate infection in lymphoid tissue before invasion of the central nervous system. Yet the plausibility of CWD spreading to people has remained elusive.

Getting data on TSEs in the USA from the government is like pulling teeth, Singeltary argues. You get it when they want you to have it, and only what they want you to have.

SNIP...FULL TEXT ;


http://www.thelancet.com/journals/laninf/article/PIIS1473309903007151/ fulltext

 

[flounder9]

price of poker goes up for those straw bred bucks

X2

It is odd that in the story the preserve owner says he has been in the cwd monitoring program for 10 years not bringing in any new deer, only raising his own stock. Most only buy semen for new bloodlines. So how did his herd become exposed to it then if thats the truth.

IF that's true, maybe semen, where is the semen coming from? or, maybe mother to offspring transmission?



PrPSc Detection and Infectivity in Semen from Scrapie-Infected Sheep

1. <LI id=contrib-1 class=contributor>Richard Rubenstein1,5, <LI id=contrib-2 class=contributor>Marie S Bulgin2, <LI id=contrib-3 class=contributor>Binggong Chang1, <LI id=contrib-4 class=contributor>Sharon Sorensen-Melson2, <LI id=contrib-5 class=contributor>Robert B Petersen3 and
2. Giuseppe LaFauci4

+ Author Affiliations

1. <LI class=aff>1 SUNY Downstate Medical Center, Brooklyn, NY, USA; <LI class=aff>2 University of Idaho, Caldwell, ID, USA; <LI class=aff>3 Case Western Reserve University, Cleveland, OH, USA;
2. 4 NYS Institute for Basic Research in Developmental Disabilities, Staten Island, NY, USA

1. ↵5 E-mail: richard.rubenstein@downstate.edu

• <LI class=received xmlns:hwp="http://schema.highwire.org/Journal">Received 13 October 2011.
• Accepted 3 February 2012.

Abstract






A scrapie-positive ewe was found in a flock that had been scrapie free for 13 years, but housed adjacent to scrapie-positive animals, separated by a wire fence. Live animal testing of the entire flock of 24 animals revealed 7 more subclinical scrapie-positive ewes. We hypothesized that they may have contracted the disease from scrapie-positive rams used for breeding four months prior, possibly through the semen. The genotypes of the ewe flock were highly scrapie-susceptible and the rams were infected with the "Caine" Scrapie Strain having a short incubation time of 4.3-14.6 mo. in sheep with 136/171 VQ/VQ and AQ/VQ genotypes. PrPSc accumulates in a variety of tissues in addition to the central nervous system. Although transmission of prion diseases, or transmissible spongiform encephalopathies, has been achieved via peripheral organ or tissue homogenates as well as by blood transfusion, neither infectivity nor PrPSc have been found in semen from scrapie-infected animals. Using serial protein misfolding cyclic amplification followed by a surround optical fiber immunoassay, we demonstrate that semen from rams infected with a short incubation time scrapie strain contains prion disease-associated seeding activity that generated PrPSc in sPMCA. Injection of the ovinized transgenic mouse line TgSShpPrP with semen from scrapie-infected sheep resulted in PrPSc seeding activity in clinical and, probably as a result of the low titer, nonclinical mouse brain. These results suggest that the transmissible agent, or at least the seeding activity, for sheep scrapie is present in semen. This may be a strain specific phenomenon.






http://vir.sgmjournals.org/content/early/2012/02/02/vir.0.038802-0.abstract







PPo3-40:




Mother to Offspring Transmission of Chronic Wasting Disease




Candace K. Mathiason, Amy V. Nalls, Kelly Anderson, Jeanette Hayes-Klug, Nicholas Haley and Edward A. Hoover Colorado State University, Department of Microbiology, Immunology and Pathology, Fort Collins, CO USA




Key words: Chronic wasting disease, vertical transmission, muntjac deer




We have developed a new cervid model in small Asian muntjac deer (Muntiacus reevesi) to study potential modes of vertical transmission of chronic wasting disease (CWD) from mother to offspring. Eight of eight (8/8) muntjac doe orally infected with CWD tested PrPCWD lymphoid positive by 4 months post infection. Six fawns were born to these CWD-infected doe. Six fawns were born to 6 CWD-infected doe; 4 of the fawns were non-viable. The viable fawns have been monitored for CWD infection by immunohistochemistry and sPMCA performed on serial tonsil and rectal lymphoid tissue biopsies. PrPCWD has been detected in one fawn as early as 40 days of age. Moreover, sPMCA performed on rectal lymphoid tissue has yield positive results on another fawn at 10 days of age. In addition, sPMCA assays have also demonstrated amplifiable prions in maternal placental (caruncule) and mammary tissue of the dam. Additional pregnancy related fluids and tissues from the doe as well as tissue from the nonviable fawns are currently being probed for the presence of CWD. In summary, we have employed the muntjac deer model, to demonstrate for the first time the transmission of CWD from mother to offspring. These studies provide the foundation to investigate the mechanisms and pathways of maternal prion transfer.





http://www.prion2010.org/bilder/prion_2010_program_latest_w_posters_4_.pdf?139&PHPSESSID=a30a38202cfec579000b77af81be3099



just pondering here. ....



kind regards,
terry

 

[iowaqdm]

Well how did the meeting go? Anyone care to share what was discussed.

 

[jclaws]

I saw fishbonker taking notes so he can tell you alot more than me. I just didn't realize how much it came down to the owner having to cooperating with the dnr. They just finished some sort of electric fence that is 3ft away from the existing high fence to prevent nose to nose contact with wild deer. The dnr paid for half and the owner paid for the other half. It sounded like the dnr was going to have to pay for the whole thing, but the landowner stepped up and paid for the other half.
Another thing that was brought up was that the deer was only in the pen for 2 hours before it was shot,And the enclosure it was shot in was only 14 acres. That would make it alot less likely( in my opinion) to have had time to transfer the disease to very many deer. What is pretty strange is that the pen is under quarantine to keep any deer from leaving the facility.But they have been bringing new deer into the facility from other non cwd facilitys. Every deer still has to be killed by jan 31st 2013.
I asked what the plan was if a deer does test positive outside the pen, and there response was that they would contact the landowners and ask them to shoot alot more deer. But it was also brought up that under the current cwd plan that i believe was wrote up in 2002, Is that if a positive test was found that the plan was to reduce the heard to 5-10% of what it is. Im guessing that would be within a 5 mile radius. They also want 300 deer harvested within that 5 mile zone to be tested, so they are working with landowners to get in touch with the local conservation officers and have them tested. Im just hoping none of the wild deer test positive.

 

[iowaqdm]

Thanks for the info.

 

[flounder9]

Iowa residents raise concern about CWD

see video ;




Iowa residents raise concern about CWD



by Monisha Bruner


Posted: 10.02.2012 at 10:25 PM





OTTUMWA, IOWA -- It’s the first formal public meeting since chronic wasting disease was found in Iowa, and residents were not too pleased about it.

Hundreds of residents attended Tuesday night’s meeting in Ottumwa, and many questioned why this was the first meeting on the topic.

They were told because the Iowa Department of Natural Resources and lawmakers had to go over several things first.

CWD was discovered in captive deer in pens in three counties: Davis County, Cerro Gordo County and Pottawattamie County.

It appears all three cases are a result of deer being transferred from a breeding pen owned by Tom Brakke in Cerro Gordo County.

The Department of Agriculture has quarantined several other pens around Iowa that have deer from Brakke's facility.

Iowa State Representative Curt Hanson of Fairfield said this was an important meeting.

“(The) responsibilities between the breeding pens and the preserves are divided,” Hanson said. “So, we need to have some openness and discussion about what's happening and (the) purpose of this meeting is to find out what's really being done, what can be done, what we need to plan for and if there needs to be different legislation enacted.

When one resident asked if Iowa needs new legislation, one official said "yes."

Iowa’s first case of CWD was found July 16, 2012.

Officials said that, as of Tuesday, there is a 3 wire electric fence around the deer that are being quarantined so they don't spread the disease to wild deer.

Officials also said all the deer will die at the pen by the end of January 2013.



http://www.heartlandconnection.com/news/story.aspx?id=808297

 

[loneranger]

Crazy to me, that if this thing is such a terrible threat,,as it sounds to be. Why are we playing around. Shut this deer farm industry down! Find another line of work. Why threaten us all?

 

[jclaws]

Crazy to me, that if this thing is such a terrible threat,,as it sounds to be. Why are we playing around. Shut this deer farm industry down! Find another line of work. Why threaten us all?

That was brought up,and it sounds like it has to go through the legislature to get that passed.

 

[magnus]

I'm not convinced that having CWD in the wild herd is that big of a deal. It's becoming so widespread in this country, I think its only a matter of time before it gets here....one way or another. Since there hasn't been much testing donefor it outside of these high-fence hunting operations, other than on occasion, how do we know its not there already? To me, EHD is a much bigger threat to our deer herd than CWD is. I've not read or heard about many deer being found dead in in CWD-positive areas. But I do know that overzealous, panic-striken state officials have ordered their officers and agents to go onto private land and "de-populate" large numbers of deer .....for what they believe is the greater good. I thnk that in retrospect, all that kind of response does is mess up the deer herd and devalue the lands of all owners in that area. My undertanding is that Iowa has similar plans if/when CWD shows up in the wild herd.....and that's what frightens me. I've heard they said last night this is NOT their planned response for Iowa but, call me cynical, I'll have to see that in writing to believe it!!

 

[ElkHunter]

Busy collecting notes and personal thoughts from several people that attended meeting. Give me a couple days.

 

[jhertel]

I think mr. brakke could handle a little loss. He owns brakke implement out of clear lake plus he has 3 deer farms!! I think its time to shut it down!