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Fox News Article on CWD

bigbuckhunter88 said:
Don't think anyone is a none believer but would love for someone to show how many deer it kills and how killing them all is the answer. And no I don't need links to 47 useless articles that I don't have the time nor want to read.
Click to expand...

I stopped arguing with ted nugent science believers...bon appetit.

SUNDAY, FEBRUARY 10, 2019

The prion-like propagation hypothesis in Alzheimer's and Parkinson's disease

https://betaamyloidcjd.blogspot.com/2019/02/the-prion-like-propagation-hypothesis.html
 
Alot to digest again but I guess this stuff is supposed to be all linked. And could be transmitted in many ways including surgical ways.
 
My phone wouldn’t play the whole thing but a cure found in Pennsylvania? Great news! If true this should be all over the next internet
 
I have read this before. A researcher claims cwd is bacteria caused. Be nice if that is true. Bacteria can be treated. Coarse bacteria can mutate and outsmart us and here we go again
 
Hope he’s right. The prions are the byproduct as it were of a bacteria. They are not the root cause. Coarse I am sure there are a room full of experts that disagree
 
I read one of his immunolabeling electron microscopy papers, since I have experience in that area. I’m not sure his results were valid, as many times the antibodies used have cross reactivity to similar proteins. He almost bases his conclusion on this fact. I’d like to see further studies.


Sent from my iPhone using Tapatalk
 
Hardwood11 said:
Terry can explain it to us in simple terms.
Click to expand...

Dr. Bastian has been trying to prove his theory since before 1997, and no one, NO OTHER scientist in the world has been able to reproduce his supposed findings. i have followed it. i wish it were true and that simple. the bs that is coming out of the media on this 'cure' is simply just that BS. THERE IS NO CURE, AND NO CURE IN THE NEAR FUTURE FOR CWD TSE PRION.

really, to me, this is like Alzheimer's, they know it transmits now, and that it is capable of being iatrogenic, and that it is a TSE Prion. you don't hear much about that. what i am saying is, why not focus on the elephant in the room, the causes, the routes, the sources, and attack that we know. show me the transmission studies.

Re-Evidence for human transmission of amyloid-β pathology and cerebral amyloid angiopathy

>>> The only tenable public line will be that "more research is required’’ <<<

>>> possibility on a transmissible prion remains open<<<


O.K., so it’s about 23 years later, so somebody please tell me, when is "more research is required’’ enough time for evaluation ?

Re-Evidence for human transmission of amyloid-β pathology and cerebral amyloid angiopathy

Nature 525, 247?250 (10 September 2015) doi:10.1038/nature15369 Received 26 April 2015 Accepted 14 August 2015 Published online 09 September 2015 Updated online 11 September 2015 Erratum (October, 2015)

snip...see full Singeltary Nature comment here;

Alzheimer's disease

let's not forget the elephant in the room. curing Alzheimer's would be a great and wonderful thing, but for starters, why not start with the obvious, lets prove the cause or causes, and then start to stop that. think iatrogenic, friendly fire, or the pass it forward mode of transmission. think medical, surgical, dental, tissue, blood, related transmission. think transmissible spongiform encephalopathy aka tse prion disease aka mad cow type disease...

Commentary: Evidence for human transmission of amyloid-β pathology and cerebral amyloid angiopathy

http://journals.plos.org/plosone/ar...notation/933cc83a-a384-45c3-b3b2-336882c30f9d

http://journals.plos.org/plosone/article/comments?id=10.1371/journal.pone.0111492

http://journals.plos.org/plosone/ar...notation/933cc83a-a384-45c3-b3b2-336882c30f9d

https://www.frontiersin.org/articles/10.3389/fnagi.2016.00005/full

Self-Propagative Replication of Ab Oligomers Suggests Potential Transmissibility in Alzheimer Disease

*** Singeltary comment PLoS ***

Alzheimer’s disease and Transmissible Spongiform Encephalopathy prion disease, Iatrogenic, what if ?

Posted by flounder on 05 Nov 2014 at 21:27 GMT

http://www.plosone.org/annotation/listThread.action?root=82860

https://web.archive.org/web/2017012...einquiry.gov.uk/files/yb/1992/11/04001001.pdf

https://web.archive.org/web/2004031...einquiry.gov.uk/files/yb/1992/12/16005001.pdf

https://web.archive.org/web/20040315075058/www.bseinquiry.gov.uk/files/yb/1992/12/16005001.pdf

re-Evidence for human transmission of amyloid-β pathology and cerebral amyloid angiopathy
Nature 525, 247?250 (10 September 2015) doi:10.1038/nature15369 Received 26 April 2015 Accepted 14 August 2015 Published online 09 September 2015 Updated online 11 September 2015 Erratum (October, 2015)
https://www.nature.com/articles/nature15369

MY next comment below, can be found at the very bottom of the above link, wait for it to load...tss

I would kindly like to comment on the Nature Paper, the Lancet reply, and the newspaper articles.

First, I applaud Nature, the Scientist and Authors of the Nature paper, for bringing this important finding to the attention of the public domain, and the media for printing said findings.

Secondly, it seems once again, politics is getting in the way possibly of more important Transmissible Spongiform Encephalopathy TSE Prion scientific findings. findings that could have great implications for human health, and great implications for the medical surgical arena. but apparently, the government peer review process, of the peer review science, tries to intervene again to water down said disturbing findings...snip see full text;

https://www.nature.com/articles/nature15369

http://web.archive.org/web/20040315...quiry.gov.uk:80/files/yb/1992/12/16005001.pdf

https://web.archive.org/web/2017012...einquiry.gov.uk/files/yb/1992/11/04001001.pdf

https://journals.plos.org/plosone/a...notation/933cc83a-a384-45c3-b3b2-336882c30f9d

please see more of this history and references there from (these blogs are for educational use, I do not advertise or make money from this. just made a promise to mom dod 12/14/97 hvCJD, never forget, and never let them forget.) human transmission of amyloid-β pathology and cerebral amyloid angiopathy, Singeltary Submission to Nature

https://betaamyloidcjd.blogspot.com/2015/10/alzheimergate-re-evidence-for-human.html

P132 Aged cattle brain displays Alzheimer’s-like pathology that can be propagated in a prionlike manner

Ines Moreno-Gonzalez (1), George Edwards III (1), Rodrigo Morales (1), Claudia Duran-Aniotz (1), Mercedes Marquez (2), Marti Pumarola (2), Claudio Soto (1)

snip...

These results may contribute to uncover a previously unsuspected etiology surrounding some cases of sporadic AD. However, the early and controversial stage of the field of prion-like transmission in non-prion diseases added to the artificial nature of the animal models utilized for these studies, indicate that extrapolation of the results to humans should not be done without further experiments.

P75 Determining transmissibility and proteome changes associated with abnormal bovine prionopathy

Dudas S (1,2), Seuberlich T (3), Czub S (1,2)
1. Canadian Food Inspection Agency, NCAD Lethbridge Laboratory, Canada 2. University of Calgary, Canada 3. University of Bern, Switzerland.

In prion diseases, it is believed that altered protein conformation encodes for different pathogenic strains. Currently 3 different strains of bovine spongiform encephalopathy (BSE) are confirmed. Diagnostic tests for BSE are able to identify animals infected with all 3 strains, however, several diagnostic laboratories have reported samples with inconclusive results which are challenging to classify. It was suggested that these may be novel strains of BSE; to determine transmissibility, brain material from index cases were inoculated into cattle.

In the first passage, cattle were intra-cranially challenged with brain homogenate from 2 Swiss animals with abnormal prionopathy. The challenged cattle incubated for 3 years and were euthanized with no clinical signs of neurologic disease. Animals were negative when tested on validated diagnostic tests but several research methods demonstrated changes in the prion conformation in these cattle, including density gradient centrifugation and immunohistochemistry. Currently, samples from the P1 animals are being tested for changes in protein levels using 2-D Fluorescence Difference Gel Electrophoresis (2D DIGE) and mass spectrometry. It is anticipated that, if a prionopathy is present, this approach should identify pathways and targets to decipher the source of altered protein conformation. In addition, a second set of cattle have been challenged with brain material from the first passage. Ideally, these cattle will be given a sufficient incubation period to provide a definitive answer to the question of transmissibility.

=====prion 2018===

https://prion2018.org/wp-content/uploads/2018/05/program.pdf

https://prion2018.org/

***however in 1 C-type challenged animal, Prion 2015 Poster Abstracts
S67 PrPsc was not detected using rapid tests for BSE.

***Subsequent testing resulted in the detection of pathologic lesion in unusual brain location and PrPsc detection by PMCA only.

*** IBNC Tauopathy or TSE Prion disease, it appears, no one is sure ***

Posted by Terry S. Singeltary Sr. on 03 Jul 2015 at 16:53 GMT

http://www.plosone.org/annotation/listThread.action?root=86610

Discussion: The C, L and H type BSE cases in Canada exhibit molecular characteristics similar to those described for classical and atypical BSE cases from Europe and Japan.

*** This supports the theory that the importation of BSE contaminated feedstuff is the source of C-type BSE in Canada.

*** It also suggests a similar cause or source for atypical BSE in these countries. ***

see page 176 of 201 pages...tss

http://www.neuroprion.org/resources/pdf_docs/conferences/prion2009/prion2009_bookofabstracts.pdf

*** Singeltary reply ; Molecular, Biochemical and Genetic Characteristics of BSE in Canada Singeltary reply;

http://www.plosone.org/annotation/l...id=635CE9094E0EA15D5362B7D7B809448C?root=7143

http://bovineprp.blogspot.com/2018/02/

and the CDC started the TSE PRION MAD COW 'ZOMBIE' crap. totally misleading, because a cwd SUB-CLINICAL perfectly healthy look cervid can be just as dangerous.

some might find interest in this...

WEDNESDAY, FEBRUARY 20, 2019

CHRONIC WASTING DISEASE CONGRESS Serial No. 107-117 May 16, 2002 Updated 2019

https://chronic-wasting-disease.blogspot.com/2019/02/chronic-wasting-disease-congress-serial.html

cdc zombie disease prion

https://blogs.cdc.gov/publichealthmatters/2011/05/preparedness-101-zombie-apocalypse/


kind regards, terry
 
MN Hunter said:
Terry,
What do you think about the latest on this ‘study’. Dr Bastian research wasn’t peer reviewed. Nobody believes him. The monkey research wasn’t peer reviewed and everyone believes it. Interesting.
https://www.deeranddeerhunting.com/...sdom/cwd-monkeys-fed-cannibal-diet-researcher
Click to expand...

Dr. Bastian has been trying to prove his theory since before 1997, and no one, NO OTHER scientist in the world has been able to reproduce his supposed findings. i have followed it. i wish it were true and that simple. the bs that is coming out of the media on this 'cure' is simply just that BS. THERE IS NO CURE, AND NO CURE IN THE NEAR FUTURE FOR CWD TSE PRION.

https://nationaldeeralliance.com/ed...vM6ADi1q0I9hbtbSVXi5MJgusoLv6VThDX-5zPRA8NtYs

https://www.qdma.com/cwd/

WEDNESDAY, FEBRUARY 20, 2019

CHRONIC WASTING DISEASE CONGRESS Serial No. 107-117 May 16, 2002 Updated 2019

https://chronic-wasting-disease.blogspot.com/2019/02/chronic-wasting-disease-congress-serial.html
 
THURSDAY, FEBRUARY 21, 2019

Arkansas CWD TSE Prion Jumps To 605 Cases To Date As Of Jan. 10, 2019

https://chronic-wasting-disease.blogspot.com/2019/02/arkansas-cwd-tse-prion-jumps-to-605.html

THURSDAY, FEBRUARY 21, 2019

Tennessee officials concerned after 183 deer test positive for CWD TSE Prion

https://chronic-wasting-disease.blogspot.com/2019/02/tennessee-officials-concerned-after-183.html

THURSDAY, FEBRUARY 21, 2019

Pennsylvania More CWD Bethel Township, Fulton County breeding farm

https://chronic-wasting-disease.blogspot.com/2019/02/pennsylvania-more-cwd-bethel-township.html

TUESDAY, FEBRUARY 19, 2019

Mississippi CWD confirmed in additional counties Mississippi Panola and Tallahatchie

https://chronic-wasting-disease.blogspot.com/2019/02/mississippi-cwd-confirmed-in-additional.html

CHRONIC WASTING DISEASE CONGRESS Serial No. 107-117 May 16, 2002
CHRONIC WASTING DISEASE

JOINT OVERSIGHT HEARING BEFORE THE SUBCOMMITTEE ON FORESTS AND FOREST HEALTH JOINT WITH THE SUBCOMMITTEE ON FISHERIES CONSERVATION, WILDLIFE AND OCEANS OF THE COMMITTEE ON RESOURCES U.S. HOUSE OF REPRESENTATIVES ONE HUNDRED SEVENTH CONGRESS SECOND SESSION

May 16, 2002

Serial No. 107-117

snip...

Mr. MCINNIS. Today, this joint Subcommittee hearing will explore an issue of immeasurable importance to the growing number of communities in wide-ranging parts of this country, the growing incidence of Chronic Wasting Disease in North America’s wild and captive deer and elk populations. In a matter of just a few months, this once parochial concern has grown into something much larger and much more insidious than anyone could have imagined or predicted.
As each day passes, this problem grows in its size, scope, and consequence. One thing becomes clear. Chronic Wasting Disease is not a Colorado problem. It is a Wisconsin problem or a Nebraska or Wyoming problem. It is a national problem and anything short of a fully integrated, systematic national assault on this simply will not do, which is precisely why we brought our group together here today.

snip...

So this is a disease that is spreading throughout the continent and it is going to require a national response as well as the efforts that are currently taking place in States like Wisconsin, Colorado, Nebraska, Wyoming, the interest they now have down in Texas and some of the neighboring States that have large white-tailed deer population and also elk.
This is a huge issue for us, Mr. Chairman, in the State of Wisconsin. I want to commend Governor McCallum and your staff and the various agencies for the rapid response that you have shown, given the early detection of CWD after the last deer hunting season. The problem that we have, though, is just a lack of information, good science in regards to what is the best response, how dangerous is this disease. We cannot close the door, quite frankly, with the paucity of scientific research that is out there right now in regards to how the disease spreads, the exposure of other livestock herds—given the importance of our dairy industry in the State, that is a big issue—and also the human health effects.

https://www.govinfo.gov/content/pkg...m8SyMICwoGXNyQcVjcinPvAw8CvIys1lEG7hxgzWplJlk

WEDNESDAY, FEBRUARY 20, 2019

CHRONIC WASTING DISEASE CONGRESS Serial No. 107-117 May 16, 2002 Updated 2019

https://chronic-wasting-disease.blogspot.com/2019/02/chronic-wasting-disease-congress-serial.html



terry
 
Terry,

Can you show me a location or quote me a paper where we see an increase in sporadic sCJD is on the rise and what the increase is.

I live in the world of science and know how things can be spun for the future of Linton gpickets and keeping careers alive. I would like to see 1 peer reviewed article looking at rates of CWD and sCJD in areas affected

Thanks
 
jkratz5 said:
Terry,

Can you show me a location or quote me a paper where we see an increase in sporadic sCJD is on the rise and what the increase is.

I live in the world of science and know how things can be spun for the future of Linton gpickets and keeping careers alive. I would like to see 1 peer reviewed article looking at rates of CWD and sCJD in areas affected

Thanks
Click to expand...


READING OVER THE PRION 2018 ABSTRACT BOOK, LOOKS LIKE THEY FOUND THAT from this study ;

P190 Human prion disease mortality rates by occurrence of chronic wasting disease in freeranging cervids, United States

Abrams JY (1), Maddox RA (1), Schonberger LB (1), Person MK (1), Appleby BS (2), Belay ED (1) (1) Centers for Disease Control and Prevention (CDC), National Center for Emerging and Zoonotic Infectious Diseases, Atlanta, GA, USA (2) Case Western Reserve University, National Prion Disease Pathology Surveillance Center (NPDPSC), Cleveland, OH, USA..

SEEMS THAT THEY FOUND Highly endemic states had a higher rate of prion disease mortality compared to non-CWD
states.

AND ANOTHER STUDY;

P172 Peripheral Neuropathy in Patients with Prion Disease

Wang H(1), Cohen M(1), Appleby BS(1,2) (1) University Hospitals Cleveland Medical Center, Cleveland, Ohio (2) National Prion Disease Pathology Surveillance Center, Cleveland, Ohio..

IN THIS STUDY, THERE WERE autopsy-proven prion cases from the National Prion Disease Pathology Surveillance Center that were diagnosed between September 2016 to March 2017,

AND

included 104 patients. SEEMS THEY FOUND THAT The most common sCJD subtype was MV1-2 (30%), followed by MM1-2 (20%),

AND

THAT The Majority of cases were male (60%), AND half of them had exposure to wild game.

snip...

see more on Prion 2017 Macaque study from Prion 2017 Conference and other updated science on cwd tse prion zoonosis below...terry

https://prion2018.org/wp-content/uploads/2018/05/program.pdf

https://prion2018.org/

here is the last i looked. remember, surveillance for human tse prion in the usa is extremely poor. misdiagnosis. and the cases of sporadic cjd are getting much younger, and also, the new/old vpspr sporadic cjd, is just that, another name for the same damn thing. move goal post in middle of game again...

sporadic cjd cases in 1999 were 65 steady slow increase with a few blips to 233 in 2017, and 2018 has not been completed yet, takes a while. body count to date seems low, but the exposure, the sub-clinical carriers, friendly fire iatrogenic pass it foward there from via, blood, tissue, medical, dental, surgical.
tse prion in livestock spreading to pigs and camels and who knows what else, tse prion strains mutating with some of the atypicals becoming more virulent. we have been extremely lucky imo to date. but with cwd now, it's a whole new ballgame, price of poker goes up, and the strains of cwd are growing. it's just a crap shoot until one comes up and spreads to humans like cwd and scrapie spread to cervid, vertical, lateral, and God help us if a strain shows up that is transmissible by contact or via air borne, which has been proven with other tse prion disease...

***> U.S.A. CJD

Tables of Cases Examined

National Prion Disease Pathology Surveillance Center Cases Examined¹

(September 18, 2018)

Year Total Referrals² Prion Disease Sporadic Familial Iatrogenic vCJD

1998 & earlier 259 157 135 20 2 0

1999 121 73 65 7 1 0

2000 145 102 90 12 0 0

2001 209 118 110 8 0 0

2002 241 144 124 18 2 0

2003 259 160 137 21 2 0

2004 315 180 163 16 0 1³

2005 328 179 157 21 1 0

2006 365 179 159 17 1 2⁴

2007 374 210 191 19 0 0

2008 384 221 205 16 0 0

2009 397 231 210 20 1 0

2010 402 246 218 28 0 0

2011 392 238 214 24 0 0

2012 413 244 221 23 0 0

2013 416 258 223 34 1 0

2014 354 208 185 21 1 1⁵

2015 402 264 244 20 0 0

2016 397 278 247 29 0 0

2017 370 263 233 19 0 0

2018 188 129 114 7 0 0

TOTAL 6734 4085⁷ 3656⁸ 400⁹ 12 4

1Listed based on the year of death or, if not available, on year of referral;

2Cases with suspected prion disease for which brain tissue was submitted;

3Disease acquired in the United Kingdom;

4Disease acquired in the United Kingdom in one case and in Saudi Arabia in the other;

5Disease possibly acquired in a Middle Eastern or Eastern European country;

6Includes 12 cases in which the diagnosis is pending, and 20 inconclusive cases;

7Includes 13 (8 from 2018) cases with type determination pending in which the diagnosis of vCJD has been excluded.

8The sporadic cases include 3560 cases of sporadic Creutzfeldt-Jakob disease (sCJD), 63 cases of Variably Protease-Sensitive Prionopathy (VPSPr) and 33 cases of sporadic Fatal Insomnia (sFI).

9Total does not include 247 Familial cases diagnosed by blood test only.


https://case.edu/medicine/pathology...es-for-professionals/tables-of-cases-examined


https://case.edu/medicine/pathology/divisions/prion-center


https://case.edu/medicine/pathology/divisions/prion-center/resources-for-professionals

UK 1990 28 sporadic cjd cases

UK 2018 131 sporadic cjd case

http://www.cjd.ed.ac.uk/sites/default/files/figs.pdf

https://creutzfeldt-jakob-disease.blogspot.com/2018/12/creutzfeldt-jakob-disease-cjd-bse.html

not to forget;

10. ZOONOTIC, ZOONOSIS, CHRONIC WASTING DISEASE CWD TRANSMISSIBLE SPONGIFORM ENCEPHALOPATHY TSE PRION AKA MAD DEER ELK DISEASE IN HUMANS, has it already happened, that should be the question...

''In particular the US data do not clearly exclude the possibility of human (sporadic or familial) TSE development due to consumption of venison. The Working Group thus recognizes a potential risk to consumers if a TSE would be present in European cervids.''

Scientific opinion on chronic wasting disease (II)

EFSA Panel on Biological Hazards (BIOHAZ) Antonia Ricci Ana Allende Declan Bolton Marianne Chemaly Robert Davies Pablo Salvador Fernández Escámez ... See all authors

First published: 17 January 2018 https://doi.org/10.2903/j.efsa.2018.5132 ;

also, see;

8. Even though human TSE‐exposure risk through consumption of game from European cervids can be assumed to be minor, if at all existing, no final conclusion can be drawn due to the overall lack of scientific data. In particular the US data do not clearly exclude the possibility of human (sporadic or familial) TSE development due to consumption of venison. The Working Group thus recognizes a potential risk to consumers if a TSE would be present in European cervids. It might be prudent considering appropriate measures to reduce such a risk, e.g. excluding tissues such as CNS and lymphoid tissues from the human food chain, which would greatly reduce any potential risk for consumers.. However, it is stressed that currently, no data regarding a risk of TSE infections from cervid products are available.

snip...

The tissue distribution of infectivity in CWD‐infected cervids is now known to extend beyond CNS and lymphoid tissues. While the removal of these specific tissues from the food chain would reduce human dietary exposure to infectivity, exclusion from the food chain of the whole carcass of any infected animal would be required to eliminate human dietary exposure.

https://efsa.onlinelibrary.wiley.com/doi/full/10.2903/j.efsa.2018.5132

zoonosis zoonotic cervid tse prion cwd to humans, preparing for the storm

***An alternative to modeling the species barrier is the cell-free conversion assay which points to CWD as the animal prion disease with the greatest zoonotic potential, after (and very much less than) BSE..116***

https://www.tandfonline.com/doi/pdf/10.4161/pri.29237

kind regards, terry
 
Terry,
I found this from the National Institute of Health. These numbers look a little different then what you have referenced. Which one is most accurate? I do not know.
This is from the National Institute of Health.

creased. These findings are consistent with other studies that indicate a relative excess of cases among females [33], [35]–[38] but a higher incidence of CJD among the male population [35], [39]. The CJD incidence rates varied regionally, with the highest rate in the Northeast region and the lowest rates in the South and West regions. The low rate in the West is of particular interest due to the longstanding presence of chronic wasting disease (CWD) among cervids in parts of the region, particularly in Colorado and Wyoming.
 
Last edited:
MN Hunter said:
Terry,
I found this from the National Institute of Health. These numbers look a little different then what you have referenced. Which one is most accurate? I do not know.
This is from the National Institute of Health.

creased. These findings are consistent with other studies that indicate a relative excess of cases among females [33], [35]–[38] but a higher incidence of CJD among the male population [35], [39]. The CJD incidence rates varied regionally, with the highest rate in the Northeast region and the lowest rates in the South and West regions. The low rate in the West is of particular interest due to the longstanding presence of chronic wasting disease (CWD) among cervids in parts of the region, particularly in Colorado and Wyomiany.
Click to expand...


what you references Sir (without source reference url???), but i found it, is 8 YEARS OLD. things change. i have given you the latest from the prion 2018 conference, and that has not even been published yet in peer review...

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2797136/


this same scientists Maddox et al, stated today in a newspaper;

"In the past couple decades, it's been found in 24 states in the country in free-ranging animals," explains CDC Epidemiologist Dr. Ryan Maddox. "When its found in new areas that means more people are potentially exposed."


Officials just discovered the disease in Tennessee in the past few months, and 183 deer have already tested positive for the prion disease.

In the past, Maddox explains, other animal prion diseases like BSE or "mad cow disease" have transmitted to humans. That disease, now known as Variant Creutzfeldt Jakob Disease, has since killed more than 200 people, mostly in the United Kingdom.

"So we do have evidence of an animal prion disease transmitting to humans," Maddox says.

A recent scientific study carries the most substantial evidence CWD could also spread to humans.

"That study does have important implications because at least the preliminary findings that have been shared did show that Chronic Wasting Disease could spread to macaques, which are genetically close to humans," he explains.

Maddox says that study, as well as many others, is ongoing, and the CDC is calling research of CWD a top priority.

"We also are conducting studies to identify if any prion diseases are happening at a higher rate in people who may be at an increased risk for being exposed to affected deer and elk meat," Maddox says.

https://www.wkrn.com/community/heal...jVKyLQ13uF18AACXtmS_ZrgR1-rRn3DT5Za0W-3v4cZ3E

and the same scientist that said this over a 17 years ago;

Subject: CWD aka MAD DEER/ELK TO HUMANS ???
Date: September 30, 2002 at 7:06 am PST
From: "Belay, Ermias"
To: Cc: "Race, Richard (NIH)" ; ; "Belay, Ermias"
Sent: Monday, September 30, 2002 9:22 AM
Subject: RE: TO CDC AND NIH - PUB MED- 3 MORE DEATHS - CWD - YOUNG HUNTERS

Dear Sir/Madam,

In the Archives of Neurology you quoted (the abstract of which was attached to your email), we did not say CWD in humans will present like variant CJD.. That assumption would be wrong. I encourage you to read the whole article and call me if you have questions or need more clarification (phone: 404-639-3091). Also, we do not claim that "no-one has ever been infected with prion disease from eating venison." Our conclusion stating that we found no strong evidence of CWD transmission to humans in the article you quoted or in any other forum is limited to the patients we investigated.
Ermias Belay, M.D. Centers for Disease Control and Prevention

-----Original Message-----

From: Sent: Sunday, September 29, 2002 10:15 AM
To: rr26k@nih.gov; rrace@niaid.nih.gov; ebb8@CDC.GOV
Subject: TO CDC AND NIH - PUB MED- 3 MORE DEATHS - CWD - YOUNG HUNTERS
Sunday, November 10, 2002 6:26 PM .......snip........end..............TSS

Thursday, April 03, 2008

A prion disease of cervids: Chronic wasting disease 2008 1: Vet Res. 2008 Apr 3;39(4):41 A prion disease of cervids: Chronic wasting disease Sigurdson CJ.

snip...

*** twenty-seven CJD patients who regularly consumed venison were reported to the Surveillance Center***,
snip... full text ;

http://chronic-wasting-disease.blogspot.com/2008/04/prion-disease-of-cervids-chronic.html

> However, to date, no CWD infections have been reported in people.

key word here is 'reported'. science has shown that CWD in humans will look like sporadic CJD. SO, how can one assume that CWD has not already transmitted to humans? they can't, and it's as simple as that. from all recorded science to date, CWD has already transmitted to humans, and it's being misdiagnosed as sporadic CJD. ...terry

*** LOOKING FOR CWD IN HUMANS AS nvCJD or as an ATYPICAL CJD, LOOKING IN ALL THE WRONG PLACES $$$ ***

*** These results would seem to suggest that CWD does indeed have zoonotic potential, at least as judged by the compatibility of CWD prions and their human PrPC target. Furthermore, extrapolation from this simple in vitro assay suggests that if zoonotic CWD occurred, it would most likely effect those of the PRNP codon 129-MM genotype and that the PrPres type would be similar to that found in the most common subtype of sCJD (MM1).***

http://www.tandfonline.com/doi/full/10.4161/pri.28124?src=recsys

http://www.tandfonline.com/doi/pdf/10.4161/pri.28124?needAccess=true

https://wwwnc.cdc.gov/eid/article/20/1/13-0858_article


kind regards, terry
 
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